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Response of a Patient to Treatment With Adenosine-5-Monophosphate

ARNOLD C. RATNER, MD; RICHARD L. DOBSON, MD

Arch Dermatol. 1964;89(4):505-514.

Abstract
There is reason to suspect that an aberration in purine metabolism may represent part of the defect of the hepatic porphyrias.

A case of hepatic cutaneous porphyria of the symptomatic type is reported. The patient was treated with adenosine-5-monophosphate for more than one year. A reduction in urine uroporphyrin excretion, as well as a decreased incidence and severity of photosensitivity reactions were noted.

Author Affiliations
PORTLAND, ORE

Present Address: Dermatology Branch, National Cancer Institute, National Institutes of Health, Public Health Service, US Department of Health, Education, and Welfare, Bethesda, Md 20014 (Dr. Ratner).

From the Division of Dermatology, University of Oregon Medical School.

Footnotes
This study was supported in part by a NIH Training Grant, 5-TI-AM-5300-3 and, in part, by the US Army Research and Development Command, Contract No. DA-49-193-MD-2184.

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CRIPPS
Arch Dermatol 1966;94:682-686.
ABSTRACT