This Article  • Full text  • PDF  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citing articles on HighWire  • Citing articles on ISI (2)  • Contact me when this article is cited  Related Content  • Related article  • Similar articles in this journal

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

Porphyria cutanea tarda (PCT) is a photodermatosis with an onset usually occurring in patients in their third decade. Although most cases are sporadic, autosomal dominant inheritance of the disorder has been reported. In most patients, a deficiency of the uroporphyrinogen III decarboxylase is noted. Skin symptoms include vesicular, bullous, and ulcerative lesions primarily on light-exposed sites due to increased skin fragility, milia on hands, hypertrichosis, and periorbital suffusion. In about 25% of the patients diabetes is found and about one third of patients show elevated plasma iron levels. Patients with PCT excrete elevated amounts of porphyrins in the urine with a characteristic pattern (increased excretion of uroporphyrins I and III and isocoproporphyrins).¹

Report of a Case

A 77-year-old man with Fitzpatrick skin type III presented in October 1997 with multiple vesicles and bullae and indolent red and partially crusted erosions on his forearms, the dorsa of his hands, and the occipital region. The lesions . . . [Full Text of this Article]

Comment

RELATED ARTICLE

UV-B Phototoxic Effects Induced by Atorvastatin
Marie-Claude Marguery, Nadia Chouini-Lalanne, Charlotte Drugeon, Agnés Gadroy, Paule Bayle, Fabiènne Journe, Jacques Bazex, and Michel D’Incan
Arch Dermatol. 2006;142(8):1082-1084.
EXTRACT | FULL TEXT  

THIS ARTICLE HAS BEEN CITED BY OTHER ARTICLES

UV-B Phototoxic Effects Induced by Atorvastatin.
Marguery et al.
Arch Dermatol 2006;142:1082-1084.
FULL TEXT