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Arch Dermatol. 2007;143(1):109-114.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

Diagnosis: Anetoderma associated with antiphospholipid antibodies.

MICROSCOPIC FINDINGS:

Hematoxylin-eosin staining showed an unremarkable epidermis and a mild superficial dermal perivascular lymphocytic infiltrate. No thrombotic phenomena were observed. Verhoeff–van Gieson staining revealed an absence of elastic fibers in the dermis.

DISCUSSION

Anetoderma, also known as macular atrophy, was first described by Jadassohn in 1892. It is an unusual disease of unknown etiology. It is clinically characterized by small, atrophic papules that herniate inward at palpation¹ and is classified as primary or secondary. Primary anetoderma develops on clinically normal skin without any preceding dermatoses and may be associated with several systemic diseases. It is broadly categorized into 2 clinical types: Schweninger-Buzzi anetoderma (no preceding erythema) and Jadassohn-Pellizari anetoderma (preceded by macular erythema or papular urticaria). Secondary anetoderma develops over other dermatoses such as acne, pilomatricoma, syphilis, and varicella.¹ Anetoderma may present as multiple round, nonfollicular, finely wrinkled patches of skin that appear sunken, atrophic, or flaccid and demonstrate inward herniation. Lesions . . . [Full Text of this Article]

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Arch Dermatol. 2007;143(1):109-114.
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