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Johathan K. Wilkin, MD
Dermatology Section McGuire Veterans Administration Center Richmond, VA 23249

Arch Dermatol. 1984;120(5):579-580.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.—

Uhlin and McCarty¹ provide an additional observation compatible with the view that unilateral dermatomal superficial telangiectasia (UDST) (or unilateral nevoid telangiectasia) represents a localized increased sensitivity of the skin to female hormones. The speculation that a "dermatomal developmental defect in which estrogen receptors are increased would explain all of the situations"^1(p228) in which UDST is seen, is presented without citations recognizing earlier original works that proposed this pathophysiologic mechanism based on clinical observations. In 1978, it was noted that "the unilateral dermatomal distribution of the superficial telangiectasia suggests a nevoid distribution of target end organs sensitive to estrogens."² In this statement, credit was given to Selmanowitz³ who observed that "if a circulating hormone induced spider angiomas in the patients presented, then the unilateral distribution of the telangiectases might have represented a nevoid predisposition of target end organs in the vessels, or supportive connective . . . [Full Text PDF of this Article]

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