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CPT Stephen J. Friedman, MC; W. P. Daniel Su, MD; John A. Doyle, MD
USA William Beaumont Army Medical Center Dermatology Service El Paso, TX 79920-0115; Mayo Clinic and Mayo Foundation 200 First St SW Rochester, MN 55905

Arch Dermatol. 1985;121(12):1484.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.—

In their recent article, Person and Longcope¹ reported no measurable estrogen or progesterone receptors in an involved lesion of a patient with generalized essential telangiectasia (GET). They suggested that GET is unrelated to the unilateral nevoid telangiectasia syndrome (unilateral dermatomal superficial telangiectasia [UDST]) because of the presence of estrogen and progesterone receptors in the latter disorder.²

Review of the literature regarding UDST reveals two groups of patients: (1) a group with the onset of telangiectasia chronologically related to conditions associated with increased estrogen states, including puberty in women, pregnancy, and alcoholism with cirrhosis, and (2) a group that does not fit into an estrogen model for the onset of the lesions, including patients in which the UDST arose at birth or during childhood before puberty.³

Uhlin and McCarty² reported elevated levels of estrogen and progesterone receptors in only involved skin of a woman with . . . [Full Text PDF of this Article]

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