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Reuven Bergman, MD; Rachel Friedman-Birnbaum, MD; Ruth Ludatscher, MD; Chaim Lichtig, MD
Department of Dermatology Rambam Medical Center PO Box 9602 Haifa 31096, Israel; Department of Pathology Rambam Medical Center

Arch Dermatol. 1987;123(9):1127-1129.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.—

Since elastosis perforans serpiginosa (EPS) was first described, it has been suggested that it is a phenomenon caused by a variety of abnormal elastic fibers. At least three etiologic types are now considered to be involved, ie, the idiopathic, the reactive, and the penicillamine-induced.^1-3 Previous ultrastructural investigations showed that the abnormal elastic fibers in the idiopathic type differ in structure from those of the penicillamine-induced type.^1,3-6 The reactive type, which is associated with different congenital disorders, mostly of the connective tissue, such as Down's syndrome, osteogenesis imperfecta, Marfan's syndrome, pseudoxanthoma elasticum, and Ehlers-Danlos syndrome (EDS), has not been as extensively studied.⁷ It remains to be demonstrated whether it is a unique structural form of EPS.

We performed an ultrastructural study in two patients with the reactive type of EPS.

Report of Cases.—CASE 1.—

A 15-year-old girl presented with osteogenesis imperfecta, type Ia, . . . [Full Text PDF of this Article]

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