This Article  • References  • Full text PDF  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citation map  • Citing articles on HighWire  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

Thomas T. Provost, MD

Arch Dermatol. 1994;130(10):1308-1310.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

PREVIOUS RESEARCH on cutaneous lupus erythematosus lesions has been directed at examining the relationship between the scarring lesions of discoid lupus erythematosus (DLE) and systemic lupus erythematosus (SLE) and the pathogenesis of the lesions of cutaneous lupus erythematosus.

The lesions of DLE generally occur as scarring isolated cutaneous lesions in the absence of systemic features. However, studies of large populations of patients with SLE indicate that approximately 15% to 20% of patients with SLE (satisfying the American College of Rheumatology criteria for SLE) also possess discoid lupus lesions.¹ In addition, it has been recognized that over time approximately 5% to 10% of patients with DLE will develop clinical features that satisfy the American College of Rheumatology diagnosis of SLE.

Previous studies by Kulick et al² have indicated that approximately 25% of patients with DLE possess anti—single-stranded DNA antibodies of the IgM isotype. Furthermore, Callen et al³ have . . . [Full Text PDF of this Article]

Author Affiliations
Department of Dermatology Blalock 920 The Johns Hopkins Medical Institute 600 N Wolfe St Baltimore, MD 21287-4915

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?