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J. Castanet, MD
University of Nice 06107 Nice Cedex 2, France

J. Ph. Lacour, MD; I. Bodokh, MD; S. Bekri, MD; J. P. Ortonne, MD
Nice, France

Arch Dermatol. 1994;130(5):664-665.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Several authors have reported the onset of porphyria cutanea tarda (PCT) in human immunodeficiency virus (HIV)—infected patients.^1-3 We and other authors^4,5 have shown a strong association between PCT and hepatitis C virus (HCV). As HIV and HCV infections have common risk factors, especially blood transfusion and intravenous drug abuse, HIV-associated PCT might be also related to HCV.

In 1990, we examined a 30-year-old man who had a history of intravenous drug use and was documented as being positive for antibody to HIV since 1987. He had no symptoms related to immunodeficiency or liver disease. He had typical skin manifestations of PCT, which was confirmed by an elevated urinary porphyrin level. Liver function test results were as follows: alanine aminotransferase, 85 U/L (normal, 5 to 40 U/L); aspartate aminotransferase, 53 U/L (normal, 5 to 30 U/L); -glutamyl transpeptidase, 93 U/L (normal, 7 to 33 U/L); and alkaline phosphatase, 62 . . . [Full Text PDF of this Article]

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