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Lloyd E. King, Jr, MD; Jane H. Park, PhD; Lauren B. Adams, MD; Nancy J. Olsen, MD

Arch Dermatol. 1995;131(5):522-524.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

REPORT OF CASE

A 35-year-old woman (patient 2 in reference 1) was referred to Vanderbilt University Medical Center, Nashville, Tenn, in February 1991 with a 6-month history of a progressive skin eruption, including malar erythema without a heliotrope eruption, Gottron's papules, and poikiloderma on her trunk and arms. She also complained of increasingly severe proximal muscle weakness. An electromyogram indicated myositis, and a muscle biopsy specimen showed changes consistent with polymyositis. Laboratory evaluation of serum muscle enzyme levels, eg, creatine kinase, lactate dehydrogenase, aspartate aminotransferase, and aldolase, showed values within the normal range. Additionally, laboratory tests showed the fluorescent antinuclear antibody titer to be 1:320, with a speckled pattern, and negative antibody titers for double-stranded DNA, ribonuclear protein, Smith antigen, SS-A, SS-B, and Jo-l.

The patient's clinical course was therapeutically managed over the next 35 months with varying dosages of prednisone, methotrexate, azathioprine, and hydroxychloroquine. The first 5 months of . . . [Full Text PDF of this Article]

Author Affiliations
Vanderbilt University (Drs King, Park, Adams, and Olsen) and Bureau of Veterans Affairs Medical Center (Dr King), Nashville, Tenn

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