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A Serial Killer or an Innocent Bystander?

Omar P. Sangüeza, MD

Arch Dermatol. 1997;133(9):1156-1157.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

During THE past few years, the Epstein-Barr virus (EBV) has emerged as a possible cause of a large number of lymphoid proliferations. The spectrum of EBV-associated lymphoid proliferations is broad and includes T-cell lymphomas,¹ B-cell lymphomas in immunosuppressed individuals,^2,3 Hodgkin disease,⁴ and CD30⁺ anaplastic large cell lymphomas.⁵ Is EBV capable of inducing all these lymphoid neoplasms, or is it just an innocent bystander in the wrong place at the wrong time? Let us analyze the possible role of EBV in the etiopathogenesis of different lymphoid proliferations.

In this issue of the ARCHIVES, Iwatsuki et al⁶ report on the clinicopathological manifestations of different types of cutaneous lymphomas associated with EBV. Based on their findings, they propose that EBV-associated cutaneous lymphoid proliferations be divided into the following 4 groups: subcutaneous lymphoma associated with hemophagocytosis, hydroa vacciniforme (HV)—like vesiculopapular eruptions, angiocentric lymphomas (ALs), and histiocytoid lymphomas associated with . . . [Full Text PDF of this Article]

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