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SAUL S. ROBINSON, M.D.

Arch Derm Syphilol. 1937;36(5):1054-1057.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

During the past few years the etiology of scleroderma has received considerable attention from European and American investigators. Among many of the possible causes, the glands of internal secretion¹ and the sympathetic nervous system² have been considered both separately and together as playing important rôles in the production of the pathologic changes noted in this disease. Osler¹ⁱ and Oliver,³ however, have summed up the actual knowledge as to the source of scleroderma in the statement that little progress has been made in determining its exact cause. Sclerodactylia has been classified as an angiotrophoneurosis related to endocrine disturbance and to dysfunction of the sympathetic nervous system.⁴ Sellei⁵ has recently suggested dysfunctions of ferment-producing organs, principally the pancreas, as etiologic agents in scleroderma and sclerodactylia and has advocated the administration of pancreatic, duodenal and gastric ferments for the relief of the vascular spasm, especially when associated . . . [Full Text PDF of this Article]

Author Affiliations
LOS ANGELES

From the Department of Dermatology, the Cedars of Lebanon Hospital.

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