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E. D. TELFORD, F.R.C.S. Professor of Surgery Emeritus, University of Manchester FLINTSHIRE, ENGLAND

Arch Derm Syphilol. 1937;36(5):952-963.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

It is evident that continued circulatory deficiency must produce changes in the nutrition of the affected area. The essential change is the development of fibrosis. On clinical grounds these complications of circulatory deficiency may be divided into two groups. In one group, which may be called the "chilblain" group, the lesions are subacute and localized and individually tend to clear up, while in a second group there is a slow persistent and relentless interstitial fibrosis such as is seen in acrosclerosis.

While in the first group it is evident that a defective venous return is the cause, the lesions in the second group appear to arise from an insufficient arterial supply.

GROUP I

The feature of the first group is the formation of subcutaneous nodules, which commonly disappear in time but occasionally break down and lead to a painful and tedious ulceration. The study of these nodules is best begun . . . [Full Text PDF of this Article]

Footnotes
Lecture delivered for the University of London at St. Mary's Hospital Medical School, Feb. 16, 1937.

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