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Preliminary Report

FRANK E. CORMIA, M.D.; BRUCE M. ESPLIN, M.D.

Arch Derm Syphilol. 1950;61(6):931-945.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

THE SOLUTION of the problems of eczema depends on the correlation of many interrelated facts. One apparent connecting link is the process of autoeczematization, which develops in many types of eczematous dermatoses. Autoeczematization, as considered here, consists in the development of scattered eczematous lesions complicating an acute eczematous process ordinarily of limited extent.

Because of the paucity of experimental work on what is felt to be a fundamental and important problem, a study has been instituted of some of the mechanisms involved. This paper is a preliminary report of such studies.

SURVEY OF THE LITERATURE

The concept of autoeczematization stems from the observations of Whitfield,¹ who postulated that a person might become sensitized to split products of autogenous tissue proteins. Whitfield described three types of reactions, one, and possibly two, of which fits into the present day concept of autoeczematization. The first consisted of widely distributed, . . . [Full Text PDF of this Article]

Author Affiliations
NEW YORK

From the Department of Medicine (Dermatology), Cornell University Medical College and the New York Hospital.

Footnotes
Read at the Sixty-Ninth Annual Meeting of the American Dermatological Association, Inc., at Hot Springs, Va., May 25, 1949.

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