This Article  • References  • Full text PDF  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citation map  • Citing articles on Web of Science (4)  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

W. M. SHAW, M.D.; E. H. MASON, M.D.; F. G. KALZ, M.D.

AMA Arch Derm Syphilol. 1952;66(2):197-203.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

THE SYNDROME of carotenemia, which was first described by von Noorden in 1907,¹ has been observed in a variety of metabolic disorders, including diabetes mellitus, chronic glomerulonephritis, myxedema, acute hepatic disease, xanthomatosis, and the ingestion of unusual quantities of vegetables rich in carotenoid pigments.

Carotene is not synthesized by man. The absorption of carotene and its biologically active form, vitamin A, from the small intestine is linked with the absorption of lipids, is enhanced by the presence of lecithin, and, in the case of carotene, is dependent on the presence of bile salts. About 30 to 50% of the ingested carotene is converted to vitamin A,² and in man both conversion and storage take place in the liver.³ Mattson and associates have demonstrated that conversion in rats takes place chiefly in the intestinal mucosa.⁴ Because carotenemia usually occurs in association with diseases . . . [Full Text PDF of this Article]

Author Affiliations
WINNIPEG, MAN., CANADA; MONTREAL, CANADA

From the Department of Medicine, Royal Victoria Hospital, Montreal, Canada.

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?