This Article  • References  • Full text PDF  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citing articles on HighWire  • Citing articles on Web of Science (12)  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

J. J. ZOON, M.D.; J. W. H. MALI, M.D.

AMA Arch Derm. 1957;75(4):573-578.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Clinical experience makes it indeed very likely that an extensive inflammation of the skin causes a repercussion on the entire organism. The cachectic and fatal end of a not unimportant number of our chronic erythroderma patients clearly points in this direction. Only rarely in these patients changes are found after death, related to the erythroderma, which by themselves may explain the fatal outcome. I would like to begin by stating that the thesis, defended by many authors, that internal disorders (metabolic diseases such as diabetes, or carcinoma, endocrine disturbances, senectitude) would be of great significance in a causative or predisposing sense in the development of erythroderma, is not acceptable in its general implications, but at most in incidental cases only. No one has ever succeeded even in making it seem plausible that an erythroderma would as a rule be the result of a primary internal disturbance. Endocrine . . . [Full Text PDF of this Article]

Author Affiliations
Utrecht, Netherlands

From the Division of Dermatology, University of Utrecht. Prof. J. J. Zoon, M.D., Director.

Footnotes
Received for publication May 7, 1956.

Read before the 14th Annual Meeting of the American Academy of Dermatology and Syphilology, Chicago, Dec. 7, 1955.

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?