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A Review and Report of a Case Treated by Chelation

SHERWYN M. WOODS, M.D.; HENRY A. PETERS, M.D.; STURE A. M. JOHNSON, M.D.

AMA Arch Derm. 1958;77(5):559-567.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Since Günther's¹ clinical study of the now-famous case of Petry, a congenital porphyric (erythropoietica of Watson²), and Hans Fischer's³ biochemical investigation of this patient in 1915, the term porphyria has grown to embrace a group of deceptively different, yet similar, syndromes. It is perhaps best regarded as a group of inborn errors of porphyrin metabolism which find expression in the adult most commonly as an acute, often fatal, abdominal-neurological disorder or, less commonly, as a more chronic photosensitive cutaneous disorder. Increasing knowledge of the biochemistry of the porphyrins and the frequent merging of one syndrome into the other have offered a perplexing clinical problem. The classifications of Günther,¹ Waldenström,⁴ Kark,⁵ and Watson² bear witness to this problem, the latter being widely accepted in this country. Watson's classification is as follows:

Porphyria Erythropoietica

Porphyria Hepatica

Acute Intermittent

Cutanea tarda

Mixed

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Author Affiliations
Madison, Wis.

Footnotes
Submitted for publication Dec. 19, 1957.

Formerly senior student in medicine, University of Wisconsin Medical School; currently intern, Philadelphia General Hospital (Dr. Woods). Assistant Professor of Neuropsychiatry, University of Wisconsin Medical School (Dr. Peters). Professor of Medicine, University of Wisconsin Medical School (Dr. Johnson).

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